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cell autonomous eion of inflammatory genes in

  • Description:Cell autonomous expression of inflammatory genes in ...Importantly, the inflammatory gene expression signature described here is cell autonomous, i.e. occurs in the absence of prototypical immune or pro-inflammatory cells, growth factors, or other inflammatory mediators.Cited by 64Publish Year 2008Author Andres Kriete, Andres Kriete, ...
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cell autonomous eion of inflammatory genes in Description

(PDF) Cell autonomous expression of inflammatory genes

Cell autonomous expression of inflammatory genes in biologically aged fibroblasts associated with elevated NF-kappaB activityA synthetic mechanogenetic gene circuit for autonomous cell autonomous eion of inflammatory genes inApr 29, 2020Mechanobiologic signals play critical roles in regulating cellular responses under both physiologic and pathologic conditions. Using a combination of synthetic biology and tissue engineering, we developed a mechanically-responsive bioartificial tissue that responds to mechanical loading to produce a pre-programmed therapeutic biologic drug. By deconstructing the signaling networks A synthetic mechanogenetic gene circuit for autonomous cell autonomous eion of inflammatory genes inApr 29, 2020Mechanobiologic signals play critical roles in regulating cellular responses under both physiologic and pathologic conditions. Using a combination of synthetic biology and tissue engineering, we developed a mechanically-responsive bioartificial tissue that responds to mechanical loading to produce a pre-programmed therapeutic biologic drug. By deconstructing the signaling networks

Anti-Inflammatory Actions of Intravenous Immunoglobulin cell autonomous eion of inflammatory genes in

The remarkable success story of the therapeutic application of pooled immunoglobulin G (IgG) preparations from thousands of donors, the so-called intravenous IgG (IVIG) therapy, to patients with a variety of hematological and immunological disorders began more than half a century ago. Since then, the use of this primary blood product has increased constantly, resulting in the serious danger of cell autonomous eion of inflammatory genes inAt the Crossroads of Inflammation and Cancer - ScienceDirectSep 17, 2004Cell-Autonomous Effects of Inflammation Pathways in Intestinal Epithelium? The previously published literature overwhelmingly supports a scenario in which inflammation promotes (rather than initiates) carcinogenesis in a non-cell-autonomous fashion.Biochimica et Biophysica Acta (BBA) - Gene Structure and cell autonomous eion of inflammatory genes inHere, we report that heavy metals, in particular zinc, can enhance the expression of the ICAM-1 gene on cells actively involved at different levels during inflammation.

Biochimica et Biophysica Acta (BBA) - Gene Structure and cell autonomous eion of inflammatory genes in

Here, we report that heavy metals, in particular zinc, can enhance the expression of the ICAM-1 gene on cells actively involved at different levels during inflammation.Cell Autonomous and Non-cell Autonomous Regulation of Sheikh et al. demonstrate that hypoxia-induced expression of KLF4 and HIF1- in specialized lung arteriole SMC progenitors is required for distal migration and smooth muscle expansion, respectively. A HIF1-/PDGF-B axis in endothelial cells non-cell autonomously regulates progenitor SMC induction, proliferation, and differentiation. The myeloid cell lineage marks SMCs.Cell autonomous expression of inflammatory genes in cell autonomous eion of inflammatory genes inImportantly, the inflammatory gene expression signature described here is cell autonomous, i.e. occurs in the absence of prototypical immune or pro-inflammatory cells, growth factors, or other inflammatory mediators.

Cell autonomous expression of inflammatory genes in cell autonomous eion of inflammatory genes in

process in response to compromised mitochondrial function. Importantly, the inflammatory gene expression signature described here is cell autonomous, i.e. occurs in the absence of prototypical immune or pro-inflammatory cells, growth factors, or other inflammatory mediators. Background Chronic inflammation associated with the aging processCell-autonomous immunity shaped human evolution cell autonomous eion of inflammatory genes inSep 09, 2020Cell-Autonomous Immunity and The Pathogen-Mediated Evolution of Humans Or How Our Prokaryotic and Single-Celled Origins Affect The Human Evolutionary Story. The Cells Free Full-Text CD28 Autonomous Signaling Up cell autonomous eion of inflammatory genes inThe immunopathogenesis of multiple sclerosis (MS) depend on the expansion of specific inflammatory T cell subsets, which are key effectors of tissue damage and demyelination. Emerging studies evidence that a reprogramming of T cell metabolism may occur in MS, thus the identification of stimulatory molecules and associated signaling pathways coordinating the metabolic processes that amplify T cell autonomous eion of inflammatory genes in

Cells Free Full-Text CD28 Autonomous Signaling Up cell autonomous eion of inflammatory genes in

The immunopathogenesis of multiple sclerosis (MS) depend on the expansion of specific inflammatory T cell subsets, which are key effectors of tissue damage and demyelination. Emerging studies evidence that a reprogramming of T cell metabolism may occur in MS, thus the identification of stimulatory molecules and associated signaling pathways coordinating the metabolic processes that amplify T cell autonomous eion of inflammatory genes inCellular and Molecular Mechanisms of Chronic Inflammation cell autonomous eion of inflammatory genes inUtilization of well-established mouse models reveals that NF-B activity has a detrimental effect on satellite cell function via non-cell-autonomous mechanisms, and that inhibition of NF-B activity and its downstream target phospholipase A2 in skeletal muscle fibers Cited by 64Publish Year 2008Author Andres Kriete, Andres Kriete, Kelli L Mayo, Kelli L Mayo, Nirupama Yalamanchili, William Beggs, Patr cell autonomous eion of inflammatory genes in"Cell autonomous expression of inflammatory genes in cell autonomous eion of inflammatory genes inBACKGROUND Chronic inflammation is a well-known corollary of the aging process and is believed to significantly contribute to morbidity and mortality of many age-associated chronic diseases. However, the mechanisms that cause age-associated inflammatory changes are not well understood. Particularly, the contribution of cell stress responses to age-associated inflammation in 'non-inflammatory cell autonomous eion of inflammatory genes in

Cited by 64Publish Year 2008Author Andres Kriete, Andres Kriete, Kelli L Mayo, Kelli L Mayo, Nirupama Yalamanchili, William Beggs, Patr cell autonomous eion of inflammatory genes in(PDF) Cell autonomous expression of inflammatory genes in cell autonomous eion of inflammatory genes in

Importantly, the inflammatory gene expression signature described here is cell autonomous, i.e. occurs in the absence of prototypical immune or pro-inflammatory cells, growth factors, or other cell autonomous eion of inflammatory genes inCited by 64Publish Year 2008Author Andres Kriete, Andres Kriete, Kelli L Mayo, Kelli L Mayo, Nirupama Yalamanchili, William Beggs, Patr cell autonomous eion of inflammatory genes inCell autonomous expression of inflammatory genes in cell autonomous eion of inflammatory genes inJul 16, 2008Importantly, the inflammatory gene expression signature described here is cell autonomous, i.e. occurs in the absence of prototypical immune or pro-inflammatory cells, growth factors, or other inflammatory mediators.Correlation of Protein and Gene Expression Profiles of cell autonomous eion of inflammatory genes inCell autonomous expression of inflammatory genes in biologically aged fibroblasts associated with elevated NF-kappaB activity 16 July 2008 Immunity & Ageing, Vol. 5, No. 1 Changes in HLA-DR expression, cytokine production and coagulation following endotoxin infusion in healthy human volunteers

DNA and Cell Biology Mary Ann Liebert, Inc., publishers

DNA and Cell Biology delivers authoritative, peer-reviewed research on all aspects of molecular and cellular biology, with a unique focus on combining mechanistic and clinical studies to drive the field forward.. DNA and Cell Biology coverage includes:. Gene Structure, Function, and Regulation. Gene regulation; Molecular mechanisms of cell activation cell autonomous eion of inflammatory genes inGenes Free Full-Text Replication Stress, DNA Damage cell autonomous eion of inflammatory genes inComplete and accurate DNA replication is essential to genome stability maintenance during cellular division. However, cells are routinely challenged by endogenous as well as exogenous agents that threaten DNA stability. DNA breaks and the activation of the DNA damage response (DDR) arising from endogenous replication stress have been observed at pre- or early stages of oncogenesis and Images of Cell Autonomous Eion of inflammatory genes in imagesCell autonomous expression of inflammatory genes in cell autonomous eion of inflammatory genes inprocess in response to compromised mitochondrial function. Importantly, the inflammatory gene expression signature described here is cell autonomous, i.e. occurs in the absence of prototypical immune or pro-inflammatory cells, growth factors, or other inflammatory mediators. Background Chronic inflammation associated with the aging process

Inflammation 2010 New Adventures of an Old Flame cell autonomous eion of inflammatory genes in

Mar 19, 2010Inflammation is an adaptive response to noxious conditions. (A) Under normal conditions, tissue-resident macrophages maintain tissue homeostasis by removing dead cells and other debris and by producing growth factors. (B) Under noxious conditions, the cellular stress response is activated and results in a cell-autonomous adaptation.Inflammation 2010 New Adventures of an Old Flame CellInflammation is an essential immune response that enables survival during infection or injury and maintains tissue homeostasis under a variety of noxious conditions. Inflammation comes at the cost of a transient decline in tissue function, which can in turn contribute Inflammatory microenvironment remodelling by tumour cells cell autonomous eion of inflammatory genes inHere, we focus on new developments illustrating the key role of tumour cell-autonomous signalling after radiotherapy. Radiotherapy-induced tumour cell micronuclei activate cytosolic nucleic acid sensor pathways, such as cyclic GMP-AMP synthase (cGAS)-stimulator of interferon genes (STING), and propagation of the resulting inflammatory signals cell autonomous eion of inflammatory genes in

Innate Immune Cells in Inflammation and Cancer Cancer cell autonomous eion of inflammatory genes in

The innate immune system has evolved in multicellular organisms to detect and respond to situations that compromise tissue homeostasis. It comprises a set of tissue-resident and circulating leukocytes primarily designed to sense pathogens and tissue damage through hardwired receptors and eliminate noxious sources by mediating inflammatory processes.Innate Immune Cells in Inflammation and Cancer Cancer cell autonomous eion of inflammatory genes inThe innate immune system has evolved in multicellular organisms to detect and respond to situations that compromise tissue homeostasis. It comprises a set of tissue-resident and circulating leukocytes primarily designed to sense pathogens and tissue damage through hardwired receptors and eliminate noxious sources by mediating inflammatory processes.JCI - Hypoxia-dependent regulation of inflammatory cell autonomous eion of inflammatory genes inJul 25, 2016Uncontrolled inflammation underpins a diverse range of diseases where effective therapy remains an unmet clinical need. Hypoxia is a prominent feature of the inflammatory microenvironment that regulates key transcription factors including HIF and NF-B in both innate and adaptive immune cells.

JCI - Inflammatory links between obesity and metabolic

Jun 01, 2011Inflammation is a coordinated response to harmful stimuli, with the goal of returning the system back to a normal baseline. The inflammatory response triggered by obesity involves many components of the classical inflammatory response to pathogens and includes systemic increases in circulating inflammatory cytokines and acute phase proteins (e.g., C-reactive protein), recruitment of Key role for microRNA in inflammatory bowel disease cell autonomous eion of inflammatory genes inMay 09, 2017Inflammatory bowel disease (IBD), including Crohn's disease and ulcerative colitis, affects almost 2 million people in the US. Although IBD is Macrophages in inflammation, repair and regeneration cell autonomous eion of inflammatory genes inIn the early inflammatory phase triggered by tissue injury, monocytes and macrophages lead inflammation by expressing pro-inflammatory cytokines and interact with other pro-inflammatory immune cells. Macrophages are activated by various DAMPs, fibrin and pro-inflammatory cytokines (

Necroptosis promotes cell-autonomous activation of cell autonomous eion of inflammatory genes in

Necroptosis, a form of regulated necrotic cell death, is mediated by receptor interacting protein 1 (RIPK1), RIPK3, and mixed lineage kinase domain-like protein (MLKL). However, the mechanism by which necroptosis promotes inflammation is still unclear. Here we report that the expression of cytokines Non-cell-autonomous control of vascular stem cell fate by cell autonomous eion of inflammatory genes inSep 30, 2008A Non-Cell-Autonomous Model of Cell Fate Determination Between the Differentiation and Maintenance of Vascular Stem Cells. Based on these data, we propose a non-cell-autonomous model of cell fate determination between the differentiation and maintenance of vascular stem cells (Fig. 4 L and M). Phloem and their neighboring cells produce and cell autonomous eion of inflammatory genes inNon-cell-autonomous control of vascular stem cell fate by cell autonomous eion of inflammatory genes inSep 30, 2008A Non-Cell-Autonomous Model of Cell Fate Determination Between the Differentiation and Maintenance of Vascular Stem Cells. Based on these data, we propose a non-cell-autonomous model of cell fate determination between the differentiation and maintenance of vascular stem cells (Fig. 4 L and M). Phloem and their neighboring cells produce and cell autonomous eion of inflammatory genes in

Senescence-Associated Secretory Phenotypes Reveal Cell cell autonomous eion of inflammatory genes in

Dec 02, 2008Oncogenic RAS is a cell-autonomous driver of cell proliferation in many cancer cells. In normal cells, however, oncogenic RAS causes genotoxic stress and senescence [ 57 , 58 ], inducing a SASP and thereby conferring complex cell-nonautonomous oncogenic activities.Some results are removed in response to a notice of local law requirement. For more information, please see here.Some results are removed in response to a notice of local law requirement. For more information, please see here.(PDF) Cell autonomous expression of inflammatory genes Cell autonomous expression of inflammatory genes in biologically aged fibroblasts associated with elevated NF-kappaB activity

Some results are removed in response to a notice of local law requirement. For more information, please see here.(PDF) Cell autonomous expression of inflammatory genes

Importantly, the inflammatory gene expression signature described here is cell autonomous, i.e. occurs in the absence of prototypical immune or pro-inflammatory cells, growth factors, or other cell autonomous eion of inflammatory genes inTargeting the ion channel TRPM7 promotes the thymic cell autonomous eion of inflammatory genes inDec 08, 2020Given the important role that regulatory T (Treg) cells play in preventing autoimmunity by suppressing the deleterious effects of effector T (Teff) cells, strategies that increase Treg cell numbers in vivo are of great interest. Mendu et al . found that loss of the channel and kinase TRPM7 in T cells led to an increase in Treg cell number in mice and protected against Teff celldependent cell autonomous eion of inflammatory genes inTargeting the ion channel TRPM7 promotes the thymic cell autonomous eion of inflammatory genes inDec 08, 2020Given the important role that regulatory T (Treg) cells play in preventing autoimmunity by suppressing the deleterious effects of effector T (Teff) cells, strategies that increase Treg cell numbers in vivo are of great interest. Mendu et al . found that loss of the channel and kinase TRPM7 in T cells led to an increase in Treg cell number in mice and protected against Teff celldependent cell autonomous eion of inflammatory genes in

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